Thank you for the kind thought and encouragement AndiBB.
This trial result is intriguing in that it suggests the body may increase COX2 in response to an omega 3 shortage.
COX2 is the substance COX blocking drugs target.
The probable consequences of this in a high omega six world would be that the conversion ability of omega six products to the inflammatory chemicals including PGE2 would be increased.
COX2 is the substance used by the omega sixes to make the omega six family of inflammatory chemicals.
So another straw in the breeze that it is a sensible risk reduction strategy to balance omega three and six intake (as well as reducing the risk of bipolar disorder).
http://www.nature.com/mp/journal/v12.../4001887a.html
Dietary n-3 PUFA deprivation alters expression of enzymes of the arachidonic and docosahexaenoic acid cascades in rat frontal cortex
J S Rao1, R N Ertley1, J C DeMar Jr1, S I Rapoport1, R P Bazinet1 and H-J Lee1
Abstract
The finding that n-3 PUFA deprivation increases cPLA2, sPLA2 and COX-2 is opposite to what has been reported after chronic administration of anti-manic agents to rats and suggests that n-3 PUFA deprivation may increase susceptibility to bipolar disorder.
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