Explanations welcome
Can someone explain what the rationale is that is being used to hold up progress?
TEN years ago when I was diagnosed, it was possible to do comparison studies in other countries that showed the effectiveness of such treatment as ovarian ablation plus "X" drug, versus standard chemotherapy for early stage bc:
http://jncimonographs.oxfordjournals...001/30/67.full
and
http://jncimonographs.oxfordjournals...expansion.html
How long will it take for us to get a comparison of either ovarian ablation and trastuzumab alone (or possibly trastuzumab plus lapatinib) for premenopausal early stage bc patients, versus standard chemotherapy, in this country?
-AlaskaAngel
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Dx 2002 age 51
bc for granny, aunt, cousin, sister, mother.
ER+/PR+/HER2+++, grade 3
IDC 1.9 cm, some DCIS, Stage 1, Grade 3
Lumpectomy, CAFx6 (no blood boosters), IMRT rads, 1 3/4 yr tamoxifen
Rads necrosis
BRCA 1 & 2 negative
Trials: Early detection OVCA; 2004 low-dose testosterone for bc survivors
Diet: Primarily vegetarian organic; metformin (no diabetes), vitamin D3
Exercise: 7 days a week, 1 hr/day
No trastuzumab, no taxane, no AI
NED
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