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				end result being less bioavailable estrogen
			 
 Hope
 here is a comment kindly sent ot me by somebody who is researching the subject in response to a question I asked.
 
 There is a post on this site (around 12/12/06) with lots of links to technical article and trials that  look at the the phytoestrogenic impact, which you might like to show to your onc.
 
 Flax is unusually high in omega 3.  I suggest you read/skim the post on the importance of omega threes and sixes on breast cancer on the articles of interest if it is a subject you are unaware of.
 
 I hope this helps
 
 RB
 
 ABSTRACT
 
 
 ".....firstly,
 flax oil does not contain any lignans as they do not dissolve in oil.
 Lignans are present in every plant food that we eat only more
 concentrated in flax and therefore commercially more viable to extract.
 There is products available on the market that do deliver specific
 quantities of flax lignans.
 
 Lignans are phytoestrogens but they are very weak and only elicit a
 very weak estrogenic response. The current research suggests that the
 advantage of this is the body detects them as estrogens (which produce a
 strong estrogenic response) and therefore produces less harmful
 estrogens (homoeostatic process) though inhibition of the aromatase
 enzymes, more SHBG to bind estrogens (for elimination from the body) and
 increases the conversion of estrogens to the 2OHE metabolism pathway
 over the 16OHE (by increasing CYP1A1 enzymes). This whole process has a
 cyclic effect with the end result being less bioavailable estrogen
 (harmful), increased 2OHE (protective), and decreased 16OHE (harmful)."
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