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I did some research on Rexin G. The info is complicated and involves understanding the interaction between Cyclin G1, a protein called ARF and another protein Called MDM2. Apparently ARF and MDM2 act against the tumour destroying P53 protein and Cyclin D1 binds to these proteins. This would at first make it seem, at least to me, that the more Cyclin D1 the cancer cells contain, the better but apparantey this isnt true.
Rexin G seems to carry an inactive Cyclin G1 gene on a virus to replace the active Cyclin G1 gene in the cancer cells. Thus when the cancer cells cyclin G1 gene is replaced by the inactive gene in
Rexin G you get the very positive results mentioned. There have been long standing remissions in a few cases of pancreatic cancer. I need to do more research to really understand how Rexin G works as I am a pharmacist not a cancer genetecist. It does look hopeful though
Paul
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