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Need help with Flaxseed Oil...
Okay Ladies,
I have been concerned with taking Flaxseed Oil becasue I am er postive and way back when (1st) dx. heard it may perk up the estrogen? I have purchased Flaxeed Oil - Solgar brand, certified organic cold pressed, in softgels (had once tried flaxseed oil on salad) did not like it. 2 softgels are1250 mg. Is it safe and much should I take - and when - does it counter work with other supplements? Thanks All, jean |
Flax seed has to be considered separately from flax oil.
Most oil does not contain lignans / phytoestrogens, but does have a high level of the mother omega three fat. The seed does have lignans / phytoestrogens as well as all sorts of other useful things. Here is a link that might help. You will see as usual views are split on flaxseed. http://www.her2support.org/vbulletin...ight=flax+seed You will find lots more if you click on the search button above and enter flax seed as your search term. I put this here for convenience as it is difficult to wade through lots sometimes and this is a particularly useful quote. Re Flax seed I have posted this before. Maybe you might want to see what your onc thinks. The comment was provided by a person doing trials into flax seed. RB "There seems to be a lot of confusion on the website below. I will attempt to answer some of the questions posted on this site: firstly, flax oil does not contain any lignans as they do not dissolve in oil. Lignans are present in every plant food that we eat only more concentrated in flax and therefore commercially more viable to extract. There is products available on the market that do deliver specific quantities of flax lignans. Lignans are phytoestrogens but they are very weak and only elicit a very weak estrogenic response. The current research suggests that the advantage of this is the body detects them as estrogens (which produce a strong estrogenic response) and therefore produces less harmful estrogens (homoeostatic process) though inhibition of the aromatase enzymes, more SHBG to bind estrogens (for elimination from the body) and increases the conversion of estrogens to the 2OHE metabolism pathway over the 16OHE (by increasing CYP1A1 enzymes). This whole process has a cyclic effect with the end result being less bioavailable estrogen (harmful), increased 2OHE (protective), and decreased 16OHE (harmful)." |
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