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Old 04-30-2009, 12:43 AM   #1
Rich66
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Bee Propolis Stops Tumors from Neurofibromatosis and Cancer

Bee Propolis Stops Tumors from Neurofibromatosis and Cancer

by Barbara Minton, Natural Health Editor
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(NaturalNews) Honeybees are some of the most amazing creatures ever created by nature. They play a vital role in much of the food supply through pollination, and they provide people with a healthy natural alternative to sugar. Some of the most interesting aspects of bees are the substances they make to use themselves. Propolis is a natural resin found in young tree buds. Bees collect it for use as a glue to seal their homes. Propolis is an exceedingly complex product that contains substances that prevent and treat diseases ranging from cancer to the common cold. Recent research has documented the ability of propolis to suppress the growth of human tumors from neurofibromatosis and cancer.

Compound from propolis halts tumor process in clinical trials

Neurofibromatosis is a genetic disorder that can produce coffee colored skin patches, but it is best known for producing tumors on nerve tissue including the nerves involved in hearing. Tumors from neurofibromatosis can grow anywhere else in the body including the brain and spinal cord, and if surgically removed, they can return. The condition of neurofibromatosis can be life threatening.

Neurofibromatosis affects one in every 3000 people. The body processes that form neurofibromatosis tumors are responsible for about 70% of human cancers.

Propolis halted neurofibromatosis tumor growth in a group of cancer patients taking part in a study by scientists at Universitaets Klinikum Eppendorf in Hamburg, Germany. Dysfunction of the NF1 or NF2 gene coding is the major cause of neurofibromatosis. Researchers had previously demonstrated that the human gene PAK1 is essential for the growth of both NF1 and NF2 tumors. Although several attempts have been made to develop anti-PAK1 drugs, none have been successful.

Since bee propolis contains anticancer ingredients caffeic acid phenethyl ester (CAPE) and artepillin C (ARC), both of which block the oncogenic PAK1 signaling pathways, its potential therapeutic effects on NF tumors were explored in vivo. It was demonstrated that a CAPE-rich extract of propolis mixed with water completely suppressed the growth of human NF1 and caused an almost complete regression of human NF2 (Schwannoma), that had been grafted in mice. The scientists stressed that although CAPE is not used clinically due to its poor bioavailability, it can be made soluble with the addition of lipids (fats). (Phytotherapy Resources, February)

In an earlier study, this German team identified CAPE's anti-cancer abilities. CAPE is a natural compound found in some foods, but is highly concentrated in bee propolis. Previously, propolis had been known only to have anti-cancer function through its profound ability to boost the immune system.

Clinical trials to test the compound on humans are ongoing. So far, cancer patients taking part have seen their tumor growth halted. The compound's effect on neurofibromatosis, melanoma and pancreatic cancer is being investigated. Although the trials are still in early stages, the majority of patients have shown no further growth of their tumors. Those in charge of the study believe that several other PAK-1 dependent diseases such as AIDS and Fragile X mental retardation syndrome can be impacted by the compound.

Propolis is effective against cancer of the larynx

Scientists in Brazil recently investigated the effects of propolis on human laryngeal epidermoid carcinoma. They incubated cells with different concentrations of bee propolis for different time periods. Then they analyzed morphology and number of viable cancerous cells. Their data showed that propolis exhibited the ability to kill cancerous cells in a dose and time dependent manner. (Evidence Based Complementary and Alternative Medicine, October 22, 2007)

Propolis keeps tumors from setting up their own blood supplies

In other recent research, researchers examined the ability of propolis components to stop tumors from developing their own blood supplies. When blood supply to a tumor is cut off, it can no longer receive nutrients to fuel its growth. Acacetin, apigenin, artepillin C, CAPE, chrysin, p-coumaric acid, galangin, kaempherol, pinocembrin, and quercetin were studied for their antioxidant activity as well.

Two of the components, CAPE and quercetin, possessed strong inhibitory effects on tube formation and endothelial cell proliferation, and coincidentally showed strong antioxidant activity. Artepillin C, galangin, and kaempferol also possessed strong ability to block tumor blood supply. Each had strong antioxidant activity although not to the degree of CAPE and quercetin. In contrast, acacetin, apigenin, and pinocembrin possessed a considerable degree of effect against blood supply formation, although they demonstrated low antioxidant activity. The scientists noted the potential for these compounds to be developed into pharmaceutical drugs for the treatment of human tumors. (Molecular Nutrition and Food Research, December 8, 2008)

Bee propolis rejuvenates the immune system

The first double-blind study of propolis involved a team of five doctors led by Professor S. Scheller in Poland, who found that propolis had the power to prolong the prime of life by stimulating the immune system to release substances that protect against cellular deterioration. In addition, propolis boosted the destruction of potentially harmful foreign bacteria and stimulated the formation of antibodies to build immunity to many diseases. This strengthening of cellular defense helps build resistance to aging and illness.

Phagocytes are the white blood cells that serve as the primary defense against bacterial infections. Their activity was found to be increased by propolis.

As people reach their 30s and 40s, the immune system generally begins to weaken, increasing susceptibility to disease. Propolis offers wide spectrum activity though its broad range of flavonoids and other cell-building compounds, such as vitamins, minerals and enzymes. Flavonoids from propolis can block the release of histamine by stabilizing cell membrane lipids. Flavonoids guard against deposits of aging lypofuscins, which is fatty pigment in the heart, brain, nerves and liver.

Here are some comments from Dr. Scheller about his findings regarding propolis as quoted in Carlson Wade's article Can Bee Propolis Rejuvenate the Immune System:

"There was an increase in general physical performance, also sexual, and above all intellectual.

"In the gastro-enterological area, we found a favorable effect on infectious changes in the mucous membranes of the stomach and intestines.

"There was an accelerated and intensified regeneration of injured tissue on traumatically or infectiously altered tissues.

"There were no unequivocally negative changes in the white blood picture or in the liver or kidney values. Among other things, this substantiates the absence of side effects of propolis as well as its non-toxicity.

"In our paper, we particularly studied the effect on the immune system and documented further positive effects of propolis on circulation, metabolism, physical well-being and infectious diseases.

"Simulate the immune system, and it is possible to control the aging process and enjoy a long and healthy life. Propolis holds the key to this form of inner rejuvenation."

Propolis is effective against a variety of bacteria and viruses

A bee hive is a busy place crammed full of bustling bodies. These conditions would make them very susceptible to bacterial and viral infections, which could destroy the hive the way the Bubonic Plague ravaged Europe in the 17th century. Yet bees are able to prevent infection by using sap from young trees that has antibiotic properties. They gather the sap, remetabolize it with their nectar secretions, and take it back to the hives. There they spread it all over the place so that every bee will brush against it and become immunized. The tree antibiotic becomes the bee antibiotic.

Studies have documented the ability of propolis against Staphylococcus aureus that causes deadly infections in hospitals. Researchers found that extracts of propolis inhibited the growth of the bacteria.

Another study documented that propolis inhibited the activity of streptococcal bacteria species that cause dental cavities. Japanese researchers reported that propolis fed laboratory rats had far fewer cavities than those given a regular diet.

Researchers in Brazil recently evaluated the antimicrobial activity of two experimental pastes containing propolis extract associated with calcium hydroxide against polymicrobial cultures collected from molars of children. The paste was effective in controlling dental infections. (Brazilian Dental Journal, 2008)

Propolis is equally effective against viral infection. Several journals have documented its ability to fight upper respiratory infections, such as those caused by the common cold and influenza viruses. It has been shown to prevent viruses from reproducing, but it must be used throughout the infection period.
About the author

Barbara is a school psychologist, a published author in the area of personal finance, a breast cancer survivor using "alternative" treatments, a born existentialist, and a student of nature and all things natural.
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Old 04-30-2009, 02:10 PM   #2
tricia keegan
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Thanks for posting this info Rich which I found very interesting.
The main reason being that I took bee propolis in tab form daily right through my chemo along with natural aloe vera as a drink mixed with some orange juice.
having read this I'm tempted to begin taking it again!
I'm hormone positive so need to look into it more as to whether it's safe, but at four years out it has'nt done me harm so far
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Dx July '05 IDC 1.9cm Triple positive 3/9 nodes positive
A/C X 4 ..Taxol/Herceptin x 12 wks then herceptin 1 yr
Rads x 36 ..oophorectomy August '06
Currently taking Arimidex..
June 2011 osteopenia/ zometa x1 yearly- stopped Zometa 2015 as Dexa show normal bone density.
Stopped Arimidex July 2014- Restarted Arimidex 2015 for a further two years on the advice of my Onc.
2014 Normal Dexa scan
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Old 04-30-2009, 05:04 PM   #3
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Maybe a swine flu battler too
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Old 04-30-2009, 05:05 PM   #4
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1: J Ethnopharmacol. 2007 Aug 15;113(1):1-14. Epub 2007 May 22. Links
Propolis and the immune system: a review.

Sforcin JM.
Department of Microbiology and Immunology, Biosciences Institute, UNESP, 18618-000 Botucatu, SP, Brazil. sforcin@ibb.unesp.br
Propolis has been used empirically for centuries and it was always mentioned as an immunomodulatory agent. In recent years, in vitro and in vivo assays provided new information concerning its mechanisms of action, thus a review dealing with propolis and the immune system became imperative. This review compiles data from our laboratory as well as from other researchers, focusing on its chemical composition and botanical sources, the seasonal effect on its composition and biological properties, its immunomodulatory and antitumor properties, considering its effects on antibody production and on different cells of the immune system, involving the innate and adaptive immune response. In vitro and in vivo assays demonstrated the modulatory action of propolis on murine peritoneal macrophages, increasing their microbicidal activity. Its stimulant action on the lytic activity of natural killer cells against tumor cells, and on antibody production was demonstrated. Propolis inhibitory effects on lymphoproliferation may be associated to its anti-inflammatory property. In immunological assays, the best results were observed when propolis was administered over a short-term to animals. Propolis antitumor property and its anticarcinogenic and antimutagenic potential are discussed. Since humans have used propolis for different purposes and propolis-containing products have been marketed, the knowledge of its properties with scientific basis is not only of academic interest but also of those who use propolis as well. This review opens a new perspective on the investigation of propolis biological properties, mainly with respect to the immune system.
PMID: 17580109 [PubMed - indexed for MEDLINE
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Old 04-30-2009, 05:31 PM   #5
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1: Molecules. 2009 Feb 13;14(2):738-54.Links
Ethanolic extract of propolis (EEP) enhances the apoptosis- inducing potential of TRAIL in cancer cells.

Szliszka E, Czuba ZP, Domino M, Mazur B, Zydowicz G, Krol W.
Chair and Department of Microbiology and Immunology, Jordana 19, 41 808 Zabrze Medical University of Silesia, Katowice, Poland.
Ethanolic extract of propolis (EEP) is one of the richest sources of phenolic acids and flavonoids. EEP and its phenolic compounds have been known for various biological activities including immunopotentiation, chemopreventive and antitumor effects. Tumor necrosis factor related apoptosis inducing ligand (TRAIL) is a naturally occurring anticancer agent that preferentially induces apoptosis in cancer cells and is not toxic toward normal cells. We examined the cytotoxic and apoptotic effect of EEP and phenolic compounds identified in propolis in combination with TRAIL on HeLa cancer cells. HeLa cells were resistant to TRAIL-induced apoptosis. Our study demonstrated that EEP and its components significantly sensitize to TRAIL induced death in cancer cells. The percentage of the apoptotic cell after exposure to 50 microg/mL EEP and 100 ng/mL TRAIL increased to 71.10 +/- 1.16%. The strongest cytotoxic effect in combination with TRAIL on HeLa cells exhibited apigenin and CAPE at the concentration of 50 microM (58.87 +/- 0.75% and 49.59 +/- 0.39%, respectively). In this report, we show for the first time that EEP markedly augmented TRAIL mediated apoptosis in cancer cells and confirmed the importance of propolis in chemoprevention of malignant tumors.
PMID: 19223822
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Old 04-30-2009, 05:34 PM   #6
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Suuppose I should include this rare case of propolis alllegedly creating a benign "tumor":
1: Eur Respir J. 2007 Dec;30(6):1227-30. Links
Pulmonary tumour with high carcinoembryonic antigen titre caused by chronic propolis aspiration.

Lin WC, Tseng YT, Chang YL, Lee YC.
Division of Thoracic Surgery, Dept of Surgery, National Taiwan University Hospital, 7 Chung San South Road, 10002, Taipei, Taiwan.
Carcinoembryonic antigen (CEA) titre elevation is sometimes found in benign diseases, such as gastro-intestinal tract inflammatory disease and chronic obstructive pulmonary disease; however, very high CEA titre is rarely encountered in benign pulmonary disease. A 36-yr-old female, who had suffered from body weight loss, was found to have high serum CEA titre (60.8 ng.mL(-1)). Image studies revealed one pulmonary tumour at the left lower lobe, satellite nodules and mediastinal lymphadenopathy. Left lower lobectomy and lymph node dissection were performed for suspicious pulmonary malignancy. The pathological examination revealed that the tumourous lesion was composed of small and fragmented foreign bodies, fibrinopurulent exudate and heavy eosinophils. The bronchial epithelium was characterised by goblet cell hyperplasia and CEA overexpression. The remaining lung parenchyma possessed similar foreign body reaction. The patient's medical history was reviewed and it was found that she had spread propolis topically on nasal mucosa as an adjuvant therapy to asthma for 6 months prior to this medical event. The CEA titre decreased after the operation to 14.2 and 7.88 ng.mL(-1) after 2 weeks and 6 months, respectively. Propolis is used widely in folk medicine but it also has strong sensitising potential. One rare case of propolis aspiration is reported with presentation mimicking lung cancer.
PMID: 18055707
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Old 05-01-2009, 03:22 AM   #7
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Thanks for posting Rich.Since a number of 'natural' plants have proved useful eg taxanes in the treatment of cancer I guess that we should
not be too surprised that another natural substance may help in the battle also.Ellie
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Old 05-01-2009, 11:32 AM   #8
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Thanks for the extra info from me too Rich, I'll definatly be looking into it more.
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Dx July '05 IDC 1.9cm Triple positive 3/9 nodes positive
A/C X 4 ..Taxol/Herceptin x 12 wks then herceptin 1 yr
Rads x 36 ..oophorectomy August '06
Currently taking Arimidex..
June 2011 osteopenia/ zometa x1 yearly- stopped Zometa 2015 as Dexa show normal bone density.
Stopped Arimidex July 2014- Restarted Arimidex 2015 for a further two years on the advice of my Onc.
2014 Normal Dexa scan
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Old 01-15-2010, 12:20 AM   #9
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Re: Bee Propolis Stops Tumors from Neurofibromatosis and Cancer

Br J Cancer. 1999 Mar;79(9-10):1340-6.
The flavonoid galangin is an inhibitor of CYP1A1 activity and an agonist/antagonist of the aryl hydrocarbon receptor.

FULL TEXT

Ciolino HP, Yeh GC.
Cellular Defense and Carcinogenesis Section, Basic Research Laboratory, Division of Basic Sciences, National Cancer Institute-Frederick Cancer Research and Development Center, NIH, MD 21702-1201, USA.
The effect of the dietary flavonoid galangin on the metabolism of 7,12-dimethylbenz[a]anthracene (DMBA), the activity of cytochrome P450 1A1 (CYP1A1), and the expression of CYP1A1 in MCF-7 human breast carcinoma cells was investigated. Galangin inhibited the catabolic breakdown of DMBA, as measured by thin-layer chromatography, in a dose-dependent manner. Galangin also inhibited the formation of DMBA-DNA adducts, and prevented DMBA-induced inhibition of cell growth. Galangin caused a potent, dose-dependent inhibition of CYP1A1 activity, as measured by ethoxyresorufin-O-deethylase activity, in intact cells and in microsomes isolated from DMBA-treated cells. Analysis of the inhibition kinetics by double-reciprocal plot demonstrated that galangin inhibited CYP1A1 activity in a noncompetitive manner. Galangin caused an increase in the level of CYP1A1 mRNA, indicating that it may be an agonist of the aryl hydrocarbon receptor, but it inhibited the induction of CYP1A1 mRNA by DMBA or by 2,3,5,7-tetrachlorodibenzo-p-dioxin (TCDD). Galangin also inhibited the DMBA- or TCDD-induced transcription of a reporter vector containing the CYP1A1 promoter. Thus, galangin is a potent inhibitor of DMBA metabolism and an agonist/antagonist of the AhR, and may prove to be an effective chemopreventive agent.

PMID: 10188874 [PubMed - indexed for MEDLINE]


or..maybe it works in another way??:



Breast Cancer Res. 2006;8(2):R17. Epub 2006 Mar 27.
Growth of a human mammary tumor cell line is blocked by galangin, a naturally occurring bioflavonoid, and is accompanied by down-regulation of cyclins D3, E, and A.

FULL TEXT

Murray TJ, Yang X, Sherr DH.
Department of Environmental Health, Boston University School of Public Health, Boston, MA, USA.
INTRODUCTION: This study was designed to determine if and how a non-toxic, naturally occurring bioflavonoid, galangin, affects proliferation of human mammary tumor cells. Our previous studies demonstrated that, in other cell types, galangin is a potent inhibitor of the aryl hydrocarbon receptor (AhR), an environmental carcinogen-responsive transcription factor implicated in mammary tumor initiation and growth control. Because some current breast cancer therapeutics are ineffective in estrogen receptor (ER) negative tumors and since the AhR may be involved in breast cancer proliferation, the effects of galangin on the proliferation of an ER-, AhRhigh line, Hs578T, were studied. METHODS: AhR expression and function in the presence or absence of galangin, a second AhR inhibitor, alpha-naphthoflavone (alpha-NF), an AhR agonist, indole-3-carbinol, and a transfected AhR repressor-encoding plasmid (FhAhRR) were studied in Hs578T cells by western blotting for nuclear (for instance, constitutively activated) AhR and by transfection of an AhR-driven reporter construct, pGudLuc. The effects of these agents on cell proliferation were studied by 3H-thymidine incorporation and by flow cytometry. The effects on cyclins implicated in mammary tumorigenesis were evaluated by western blotting. RESULTS: Hs578T cells were shown to express high levels of constitutively active AhR. Constitutive and environmental chemical-induced AhR activity was profoundly suppressed by galangin as was cell proliferation. However, the failure of alpha-NF or FhAhRR transfection to block proliferation indicated that galangin-mediated AhR inhibition was either insufficient or unrelated to its ability to significantly block cell proliferation at therapeutically relevant doses (IC50 = 11 microM). Galangin inhibited transition of cells from the G0/G1 to the S phases of cell growth, likely through the nearly total elimination of cyclin D3. Expression of cyclins A and E was also suppressed.

CONCLUSION: Galangin is a strong inhibitor of Hs578T cell proliferation that likely mediates this effect through a relatively unique mechanism, suppression of cyclin D3, and not through the AhR. The results suggest that this non-toxic bioflavonoid may be useful as a chemotherapeutic, particularly in combination with agents that target other components of the tumor cell cycle and in situations where estrogen receptor-specific therapeutics are ineffective.

PMID: 16569260 [PubMed - indexed for MEDLINE]



Galangin: http://www.raysahelian.com/galangin.html
Quote:
Galangin, a member of the flavonol class of flavonoid, is present in high concentrations in medicinal plants (e.g. Alpinia officinarum) and propolis, a natural beehive product. Results from in vitro and in vivo studies indicate that galangin with anti-oxidative and free radical scavenging activities is capable of modulating enzyme activities and suppressing the genotoxicity of chemicals.

Mol Pharmacol. 2000 Sep;58(3):515-25.
The bioflavonoid galangin blocks aryl hydrocarbon receptor activation and polycyclic aromatic hydrocarbon-induced pre-B cell apoptosis.

Quadri SA, Qadri AN, Hahn ME, Mann KK, Sherr DH.
Department of Environmental Health, Boston University School of Public Health, Boston, Massachusetts 02118, USA.
Bioflavonoids are plant compounds touted for their potential to treat or prevent several diseases including cancers induced by common environmental chemicals. Much of the biologic activity of one such class of pollutants, polycyclic aromatic hydrocarbons (PAH), is mediated by the aryl hydrocarbon receptor/transcription factor (AhR). For example, the AhR regulates PAH immunotoxicity that manifests as pre-B cell apoptosis in models of B cell development. Because bioflavonoids block PAH-induced cell transformation and are structurally similar to AhR ligands, it was postulated that some of them would suppress PAH-induced, AhR-dependent immunotoxicity, possibly through a direct AhR blockade. This hypothesis was tested using a model of B cell development in which pre-B cells are cultured with and are dependent on bone marrow stromal or hepatic parenchymal cell monolayers. Of seven bioflavonoids screened, galangin (3,5,7-trihydroxyflavone) blocked PAH-induced but not C(2)-ceramide- or H(2)O(2)-induced pre-B cell apoptosis. Because galangin blocked AhR-dependent reporter gene expression, AhR complex-DNA binding, and AhR nuclear translocation, inhibition of a relatively early step in AhR signaling was implicated. This hypothesis was supported by the ability of galangin to bind the AhR and stabilize AhR-90-kDa heat shock protein complexes in the presence of AhR agonists. These studies demonstrate the utility of pre-B cell culture systems in identifying compounds capable of blocking PAH immunotoxicity, define at least one mechanism of galangin activity (i.e., repression of AhR activation), and motivate the use of this and similar dietary bioflavonoids as relatively nontoxic inhibitors of AhR agonist activity and as pharmacologic agents with which to dissect AhR signaling pathways.

PMID: 10953044 [PubMed - indexed for MEDLINE]



Quote:
Known ligands of the AhR are, for the most part, man-made chemicals; natural ligands have been postulated but, with the exception of indolo[2,3]-carbazole (Jellinck et al, 1993), remain unidentified. We have hypothesized elsewhere (Ciolino et al, 1998) that dietary polyphenolic compounds are natural ligands of the AhR and galangin appears to be such a compound. These experiments demonstrate that galangin inhibits carcinogen activation in MCF-7 cells at two levels: through direct inhibition of CYP1A1 enzyme activity and by inhibiting the increase in CYP1A1 transcription caused by AhR ligands. There is no study, to our knowledge, on the chemopreventive effect of galangin in
animal models of carcinogenesis. Based on our data, galangin may be a promising candidate for in vivo chemoprevention studies.
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Old 01-15-2010, 07:48 PM   #10
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Re: Bee Propolis Stops Tumors from Neurofibromatosis and Cancer

Rich,

This last post looks interesting for E.R. + gals like Tricia and me, but I am going cross-eyed trying to read it. Needs some indenting for our estrogen deprived brains!
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Old 01-16-2010, 10:38 AM   #11
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Re: Bee Propolis Stops Tumors from Neurofibromatosis and Cancer

Thank you for posting this Rich. I used to take propolis, but forgot about it for awhile. My mom was a bee keeper and quite enthusiastic about all things natural.

Keep the info coming!
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