Iodine deficiency ! - falling intakes - goitregens - competition bromine and fluorine

[dauntricastewart]

The Deficiency of Iodine in Asia is not so much prominent, but in the West the percentage of Deficiency of Iodine is higher which is major factor causing diseases related Breast.

[R.B.]

More sources of flouride from Flouride Alert - I have copied and pasted some of the content below, but it is worth visiting the site - there is more mind numbing information on this page in relation to developmental issues - flouride can both be used to kill bugs on foods and fumigate warehouses to kill bugs in them - apparently in the US it is not obligatory to move food when fumigating warehouses which where relevant could result in very high fluoride loads in food twice a year! - so you really do not know what little extras you might be getting this Thanksgiving or Christmas

http://fluoridealert.org/issues/sources/f-pesticides/

Pesticides

Due to its high toxicity, fluoride has long been used as a pesticide. In the United States, there are currently two fluoride-based pesticides that are allowed to be sprayed on food. These are: cryolite and sulfuryl fluoride.

2) According to data from the USDA (2005), the average fluoride levels in grape products are as follows:

White grape juice = 2.13 ppm
White wine = 2.02 ppm
Red wine = 1.05 ppm
Raisins = 2.34 ppm

3) Many juice drinks that are not labeled as “grape juice” use grape juice as a filler ingredient. The use of cryolite thus contaminates many juices with fluoride.

4) Cryolite is also allowed to be added to the following products (although it is unclear how many producers actually do so, and what the resulting fluoride levels are):

Apricot, Broccoli, Brussels Sprout, Cabbage, Cauliflower, Citrus fruit, Collards, Eggplant, Kale, Kiwifruit, Kohlrabi, Lettuce, Melon, Nectarine, Peach, Pepper, Plum, Pumpkin, Squash (summer & winter), Tomato, and a number of Berries (Blackberry, Blueberry (huckleberry) Boysenberry, Cranberry, Dewberry, Loganberry, Raspberry, Strawberry, Youngberry).


Direct Fumigation of Food

6) The EPA also allows food processors to use sulfuryl fluoride as a direct fumigant of certain foods. This means that food processors can purposely spray sulfuryl fluoride directly onto certain foods.

7) Unlike structural fumigation (which takes place once or twice a year), direct fumigation is a routinely performed procedure. Thus, foods that can be directly fumigated with sulfuryl fluoride will consistently have elevated fluoride levels.

8) According to EPA’s estimates, some of the foods that will be most commonly fumigated are cocoa powder, dried beans, walnuts and dried fruits.

9) EPA estimates that, if the current regulations are not rescinded, 100% of cocoa powder, 100% of dried beans, 99% of walnuts, 69% of dried fruits, 10% of walnuts, 10% of tree nuts, and 3% of rice will be fumigated.

10) When fumigated the average fluoride levels in fumigated food is:

Brown rice = 12.5 ppm
Cocoa powder = 8.4 ppm
Almonds = 5.3 ppm
Tree nuts = 5.3 ppm
Dried beans = 4.5 ppm
White rice = 4.5 ppm
Walnuts = 2.4 ppm
Dried fruits = 1 ppm

[R.B.]

And something I had not checked before - does fluoride cross the placenta - yes - http://www.ncbi.nlm.nih.gov/pubmed/2182701 - which has scary potential implications.

The implications would be greater in those who are at risk of iodine deficiency and insufficiency in pregnancy.

Might it affect fetal brain development either directly or by inhibition of thyroid function and so iodine metabolism - Fluoride Alert saved me the trouble of looking for data - based on fetal autopsies high fluoride intake is associated with serious developmental abnormalities .

Some abstracts from the link below which is well worth a visit - truly an I want to scream moment - if mitochondria do not work everything is affected.

This data was of course related to high flouride intake, but as already discussed the uptake of flouride is altered by many factors including the form in which it is ingested, wider mineral uptake and availability, so the questions as to whether those who have intakes from water, food, drugs, excessive toothpaste, low mineral and iodine intake and other risks that may be additive are at higher risk of non optimal fetal development is as yet unanswered.



http://fluoridealert.org/studies/brain05/



The human placenta does not prevent the passage of fluoride from a pregnant mother’s bloodstream to the fetus. As a result, a fetus can be harmed by fluoride ingested pregnancy. Based on research from China, the fetal brain is one of the organs susceptible to fluoride poisoning.

As highlighted by the excerpts below, three Chinese studies have investigated fluoride’s effect on the fetal brain and each has found evidence of significant neurological damage, including neuronal degeneration and reduced levels of neurotransmitters such as norepinephrine. As noted by Yu (1996), “when norepinephrine levels drop the ability to maintain an appropriate state of activation in the central nervous system is weakened.” Studies of fluoride-treated animals have reported similar effects, including lower levels of norepinephrine. (Kaur 2009; Li 1994).

The following study involves the same fetal tissue that was examined in the Yu (1996) and Dong (1993) studies. Rather than investigating fluoride’s effect on the brain, however, this study examined fluoride’s effect on the ultrastructure of cells in several tissues in the body, including from the thyroid gland. As can be seen in the following description, the fetuses from the fluoride-exposed women were experiencing a systemic toxic effect.

Conclusions: Fluoride damage to cell structures was multifaceted. Cell membranes, mitochondria, rough endoplasmic reticulum, and nuclear membranes could all be damaged at the time of fluorosis.”


another study said

These changes indicate that fluoride can retard the growth and division of cells in the cerebral cortex. Fewer mitochondria, microtubules, and vesicles within the synapses could lead to fewer connections between neurons and abnormal synaptic function, influencing the intellectual development after birth. These questions await further research.”
SOURCE: Han H, et al. (1989). Effects of fluorine on the human fetus. Chinese Journal of Control of Endemic Diseases 4:136-138. [See study]

[R.B.]

Does the placenta reduce the passage of fluoride to the baby? It might this paper asked that question and observed lower fluoride concentrations of the fetus side of the placenta to the maternal side.

Could western diet alter placental function, permeability etc; I would be surprised if it did not.

So as ever things are rarely simple.

http://njirm.pbworks.com/f/3Role+of+...t+flurosis.pdf

[R.B.]

Nitrates in water and thyroid function. There are several papers suggesting that nitrate pollution of well water may affect thyroid function. This is the biggest effect I have seen. I do not have access to the full paper but the potential implications are depressing.

http://www.ncbi.nlm.nih.gov/pubmed/17447577

Arch Environ Occup Health. 2005 Nov-Dec;60(6):317-9.
Iodine status of children living in areas with high nitrate levels in water.
Gatseva PD1, Argirova MD.
Author information
Abstract

Several researchers have suggested a possible relationship between nitrate intake and the development of goiter in children. The present cross-sectional study included schoolchildren between the ages of 11 and 14 years from 2 villages in Bulgaria with high and low nitrate levels in drinking water. The comparison between the median urinary iodine levels of the total number of exposed (179.0 microg/l) and nonexposed (202.50 microg/l) children showed statistically significant differences. The relative risk for the children exposed to high nitrate levels in drinking water, expressed as the odds ratio, was 8.145 (95% confidence interval = 1.67-39.67). The authors considered this to be very significant. They found a statistically significant difference for the prevalence of goiter among the exposed and nonexposed children. The results of the study confirmed the role of high nitrate levels in drinking water as a health risk factor for thyroid dysfunction.