possible connection?
The adiponectin article is interesting. The way it was written was a bit confusing:
"shows how low levels of a protein hormone called adiponectin or Acrp30 may help to trigger the disease."
At first it sounded like staying fat to produce more adiponectin for release would make sense (because it would seem there would be no adiponectin released at all if one stayed thin).
I wonder if this protein is the missing piece that explains at least in part why exercise works; one eats, increasing metabolism, then exercises, increasing anabolism (and catabolism), and along with it, the release of adiponectin?
Maybe this also explains why those who were thin to begin with also get bc, if they are small eaters and also small exercisers?
In addition, I do think exercise also causes the body to naturally flush out toxins, since it increases the need for fluids and pushes fluids through the body. Even though I don't always enjoy exercising I do suspect it helps quite a lot to prevent cancer and cancer recurrence.
A.A.
P.S. Another helpful mechanism with exercise could be that through the release of endorphins, etc. there is a reduction of the release of the stress substances mentioned in the other article posted?
Last edited by AlaskaAngel; 01-30-2008 at 11:17 AM..
Reason: anabolism instead of catabolism
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