Do you have a link to the original summary?
They were all on an androgen inhibitor. What androgens does this block, as I can foresee potential areas where intervention in the androgen pathways would affect fat conversion etc.
Interesting but runs against most summaries I read. See examples below
http://www.ncbi.nlm.nih.gov/pmc/articles/PMC2374335/
Reduced prostate cancer risk was associated with high erythrocyte phosphatidylcholine levels of EPA (multivariate relative risk = 0.59; 95% confidence interval 0.37–0.95, upper vs lowest quartile) and DHA (multivariate relative risk = 0.62; 95% confidence interval 0.39–0.98, upper vs lowest quartile). These analyses support evidence from in vitro experiments for a reduced risk of prostate cancer associated with dietary fish oils, possibly acting via inhibition of arachidonic acid-derived eicosanoid biosynthesis. © 1999 Cancer Research Campaign
http://cebp.aacrjournals.org/content/12/1/64.short
Each additional daily intake of 0.5 g of marine fatty acid from food was associated with a 24% decreased risk of metastatic cancer. We found that men with high consumption of fish had a lower risk of prostate cancer, especially for metastatic cancer. Marine fatty acids may account for part of the effect, but other factors in fish may also play a role.
http://www.nrjournal.com/article/S02...003-0/abstract
In conclusion, our findings suggest that a high dietary ratio of n-6/n-3 fatty acids may increase the risk of overall prostate cancer among white men and possibly increase the risk of high-grade prostate cancer among all men.